Cellular senescence is a stable cell proliferation arrest induced by a variety of stresses
including telomere shortening, oncogene activation and oxidative stress. This process plays
a crucial role in many physiopathological contexts, especially during aging when cellular
senescence favors development of age-related diseases, shortening lifespan. However, the
molecular and cellular mechanisms controlling senescence are still a matter of active
research. In the last decade, there has been emerging literature indicating a key …